Informal Piece .pdf

This informal piece is being written to help give a better understanding on how exposed
we are to aluminum and formaldehyde. Many people are unaware of how exposed they are to
these chemicals on a daily basis. Most are uncertain of products they are in, food/drink that they
are in, or general exposure. I am going to give you three key examples on each substance. I used
credited scientific journals and complied their results to give you understanding of the outcomes
that occurred. The first three I explain will be on the element formaldehyde, than another three
on aluminum.
The first study I chose to use was research done by Yulia B Monakhova. She wrote a
study on the margin of exposure to formaldehyde in alcoholic beverages. She found that though
there were no carcinogenicity after oral consumption the rats displayed thickened and raised
ridges of the forestomach, irregular mucosal thickenings, and many histopathological gastric
changes. Those changes consisted of papillary epithelial hyperplasia, hyperkeratosis, focal
ulceration, and focal chronic atrophic gastritis. The papillary epithelial hyperplasia may be
precarcinogenic lesions. She also used results from a 104 week test based on formaldehyde
levels in drinking water. This showed that treatment with the substance in water resulted in an
increase of malignant tumors and showed specific carcinogenic effects on various organs and
tissues.
The next study I chose to summarize was done by Yuchao Zhang. His study was on bone
marrow injury induced by oxidative stress in mice from inhalation exposure to formaldehyde. All
conclusive results showed that the effects of the formaldehyde on the blood count significantly
changed the numbers. The white cell counts went down by a total of 43%, lymphocytes
decreased b 39%, the red blood cells count was decreased by 17%, and the number of platelets

increased b a total of 109%. It was concluded that the all around exposure induced bone marrow
toxicity, oxidative stress, inflammation & consequential apoptosis.
The final study I chose to use on formaldehyde was one conducted to test the effects on
workers who have occupational exposure by İnci, M., Zararsız. This found not only a significant
increase in the incidence of rental cancer with the employees, but also that acute tubular necrosis
caused by edematous obstruction occurred frequently. Also acute renal failure was known to be
caused from using intravesicle formalin to treat chronic hemorrhagic cystitis. He was also able to
find from a study of alcoholic patients that when menthol was metabolized into formaldehyde it
was causing tubular necrosis in many patients.
When searching articles on aluminum I decided to use a study done by Crisponi, G on
testing the meaning of aluminum exposure on human health and aluminum related diseases.
When this study concluded the results found that there aluminum is a type of component that
poses a risk of exposure to humans with xenobiotics and contaminants. It also showed that
newborns are at risk of aluminum toxicity in the throughout the perinatal period, childhood, and
adulthood as well.
Seneff, S. also chose to do a study relating to aluminum. He chose to research autistic
symptoms relation to aluminum and acetaminophen. His results concluded that neuoronal
damage due to the aluminum penetration into the nervous system was shown to be a possible
significant factor in autism. The fact that autism rates rose readily with the significant increases
in aluminum vaccines is highly suggestive to the evidence he compiled and presented.
The final study I will share with you was one done by Wong, W. K. on the dietary
exposure of aluminum to the residents of Hong Kong. This study found that out of 256 samples

of food that were tested 97% of them found contained aluminum levels above the limit detection
which is 0.5mg/kg. It was found that the average exposure to aluminum for an adult on a weekly
basis in Hong Kong found to be estimated at 0.60 mg/kg.